What is transient amnesia?

Transient global amnesia (TGA) is a sudden, temporary interruption of short-term memory. Although patients may be disoriented, not know where they are or be confused about time, they are otherwise alert, attentive and have normal thinking abilities.

What is the difference between a TIA and transient global amnesia?

While transient decrease in blood flow (a transient ischemic attack or “TIA”) to the hippocampus can mimic TGA, TGA usually lasts longer than a typical transient ischemic attack.

Can transient global amnesia last for days?

Transient global amnesia typically lasts for hours, although its duration may range from minutes to days.

Can transient global amnesia be permanent?

There is no specific treatment for transient global amnesia (TGA). Fortunately, this condition resolves on its own, typically within hours of onset. Most people with TGA do not experience repeat episodes.

What can cause retrograde amnesia?

Retrograde amnesia is caused by damage to the memory-storage areas of the brain, in various brain regions. This type of damage can result from a traumatic injury, a serious illness, a seizure or stroke, or a degenerative brain disease.

What part of the brain is affected by anterograde amnesia?

Amnesia can result from damage to brain structures that form the limbic system, which controls your emotions and memories. These structures include the thalamus, which lies deep within the center of your brain, and the hippocampal formations, which are situated within the temporal lobes of your brain.

Can retrograde amnesia be cured?

While there is no actual cure for retrograde amnesia, the following treatments are likely to be prescribed by a medical professional: Jogging of memory, undertaken by exposing patients to past experiences in the hope of speeding up the recovery time of the brain.

What does retrograde amnesia mean?

Retrograde amnesia. Retrograde amnesia (RA) is a loss of memory-access to events that occurred, or information that was learned, before an injury or the onset of a disease. It tends to negatively affect episodic, autobiographical, and declarative memory while usually keeping procedural memory intact with no difficulty for learning new knowledge.