How do you treat post-extubation stridor?
How do you treat post-extubation stridor?
The preferential treatment of postextubation laryngeal edema consists of intravenous or nebulized corticosteroids combined with nebulized epinephrine, although no data on the optimal treatment algorithm are available. In the presence of respiratory failure, reintubation should be performed without delay.
What is post-extubation stridor?
Post-extubation stridor is the presence inspiratory noise post-extubation indicated narrowing of the airway (can be supraglottic, but usually glottic and infraglottic) ETT can cause laryngeal oedema and ulceration as well as at the site where the cuff abuts the trachea.
What drug is used to treat post-extubation stridor?
Corticosteroids for the prevention and treatment of post-extubation stridor in neonates, children and adults.
What causes stridor after extubation?
The risk of post-extubation stridor is high especially if the patient has been intubated for more than 36 hours, with other risk factors being intubated state for more than 6 days, female gender, large for size endotracheal tubes, traumatic intubation, lack of muscle relaxant used during intubation and reintubation …
What is the risk of post extubation stridor?
SUMMARY Post-extubation stridor secondary to laryngeal edema may occur in up to 37% of extubated patients and is associated with increase cost, morbidity and mortality. The risk of laryngeal edema may be evaluated by the cuff leak test.
How is post extubation stridor secondary to laryngeal edema diagnosed?
Post-extubation stridor secondary to laryngeal edema may occur in up to 37% of extubated patients and is associated with increase cost, morbidity and mortality. The risk of laryngeal edema may be evaluated by the cuff leak test.
What should I do if I have postoperative stridor?
Cool and humidified mist ameliorates postextubation stridor by reducing mucosal edema. It is recommended for mild cases when only stridor is present.
Which is a clinical marker of postextubation stridor?
The decreased airway lumen results in an increase of air flow velocity, leading to postextubation stridor (PES), which is a clinical marker of relevant PLE.