Is Giardia A protozoa?

Giardia duodenalis is a protozoan flagellate (Diplomonadida). This protozoan was initially named Cercomonas intestinalis by Lambl in 1859.

Can you have chronic Giardia?

Some people develop a chronic form of Giardia that lasts even longer, although symptoms tend to become less severe over time. Nutritional problems — Giardia can also interfere with the way the body absorbs nutrients, so it can cause vitamin deficiencies and other nutritional problems.

What is the symptoms of flagellates Giardia lamblia?

Acute giardiasis develops after an incubation period of 1 to 14 days (average of 7 days) and usually lasts 1 to 3 weeks. Symptoms include diarrhea, abdominal pain, bloating, nausea, and vomiting.

Is Giardia lamblia commensal?

Giardia: both a harmless commensal and a devastating pathogen.

What are the signs and symptoms of giardiasis?

The spectrum varies from asymptomatic carriage to severe diarrhea and malabsorption. Acute giardiasis develops after an incubation period of 1 to 14 days (average of 7 days) and usually lasts 1 to 3 weeks. Symptoms include diarrhea, abdominal pain, bloating, nausea, and vomiting.

How is Lamblia intestinalis related to giardiasis?

Giardiasis causes Lamblia intestinalis (Giardia lamblia), which belongs to the sub-kingdom of Protozoa, the subtype Mastigophora, the order Diplomonadida, the family Hexamitidae. In the cycle of development of the simplest, two stages are distinguished: the vegetative form and the cyst.

What are the signs and symptoms of lambliasis?

The acute period lasts usually several days, after which the lambliasis often passes into a subacute or chronic stage with short-term exacerbations in the form of a loose stool and bloating, slimming, increased fatigue. The most common symptoms of giardiasis with primary infection are nausea, anorexia, bloating and rumbling in the abdomen.

Is there a detectable enterotoxin in Giardia lamblia?

Isolates of Giardia lamblia from W.B. and 3 other patients did not produce detectable enterotoxin in four different assays. W.B. had normal levels of circulating immunoglobulins, detectable intestinal immunoglobulin A, circulating immunoglobulin G anti-Giardia lamblia antibodies, and lymphocyte responsiveness to solubilized Giar­